Medical prompt boxes

After this increase, is pancreas increasing levels of insulin further more or stop? why what are the 1- experimental 2- measurements 3- trials evidences that prove (confirm) that.
very important question that needs detailed explanation, and deep insightful mechanisms
why cells inside body don't (do not) respond to internal insulin, but the cells respond to external (injected) insulin from outside?
also, explain why it overrides internal defensive mechanisms for insulin resistance?
Also, from other different perspectives explain these processes in terms of bio-chemistry, physiological pathways chemistry and chemical reaction, effects of genes, roles of neural system [CNS, PNS] and deep thinking with logical combination.

Big deep analysis about nutrients, diet and life-style?
Tell me details about different scenarios from eating chicken and eggs

Go deeply regarding constituent and genetic material , and using hormones to make chicken grows faster and larger

Talk about genetic material in eggs, can it harm the people?

Why eggs make allergy for many people?

What are effects for heat on eggs and difference between eating rae egg and semi- cooked egg and full cooked eggs

pros and cons of legumes
Why sometimes good to prevent legumes, when? How? Why?
How to get beneficial fibers for normal flora and microbiota?
What are cause for microbiota imbalance
What are different internal factors affect glucose rather than insulin (hormones- neurotransmitters- transporters-signals)? How to get benefit from them to treat diabetic patients without insulin?

What about eating greens (Vegetables and other green nutrients)
pros and cons
Why some doctors warn from it?

Effect of pure honey or organic honey for management.
How to use it for treatment for different cases?
---
What about using of oils and butters High amount use ((benefit for whom and bad for whom) and low amount use (benefit for whom and bad for whom with trusted experimental and people experience evidence? When it is recommended to prevent t oil and when to minimize it and when to increase it (Talk about different types of oils and different types of cases to make huge variety, and do the same with butter for different types of oils and different types of cases to make huge variety
and Use of Syrian sunflower seeds, pumpkin seeds, peanut seeds, and chickpeas?
High amount proper for whom and not proper for whom? When they are recommended to prevent them when to minimize them?
After that for last section:
Give all strange nutritional facts and new research results and updated effective notes
---
Requirements when you search
1- All notes must be supported with valid real references and people experiences and the most important evidences are people trials with explanation and analysis of their trials.

2- Mention different reasons and explain in deep way with mechanisms, and mention different cases and scenarios with explain differences with reason and evidences.

3-Make wide variety of date and different variations and whole big comprehensive overview for different types and points

4- Use second type of evidences for all notes: which is to collect other similar patterns examples like it or have similar pattern so you support your argument.

5-The deep research must be extensive and valuable and have all mentioned requirements for all questions with expansion and verify you meet all needs.

After this increase, is pancreas increasing levels of insulin further more or stop? Why

what are the 1-experimental 2-measurements 3-trials evidences that prove (confirm) that.
very important question that needs detailed explanation, and deep insightful mechanisms

why cells inside body don’t (do not) respond to internal insulin, but the cells respond to external (injected) insulin from outside?

also, explain why it overrides internal defensive mechanisms for insulin resistance?
Also, from other different perspectives explain these processes in terms of bio-chemistry, physiological pathways chemistry and chemical reaction, effects of genes, roles of neural system [CNS, PNS] and deep thinking with logical combination.

إذا كان السكر يذوب في الدم، وارتفاعُهُ في الجسم مضر
كيف يذوب وكيف تتكون glycated product ؟
لقد وجدنا حالات واقعية يترسب؟ ولماذا يترسب؟

سكر الدم عندها مرتفع ومع منع بعض المغذيات كالدقيق الأبيض والسكر والمعكرونة والمعجنات والدجاج
قد تحسنت والتأمت (تعافت) جروح القدم وتعافى من القدم السكري بعد وقف الدواء وبعض أنواع الأكل والاستمرار في أكل السكريات (البطاطس - الأرز العصير - الشوكولاتة = العسل - التمر) = علاج وتحسن

--------------------
Why tumor happens? What are benefits or compensation motive of cells to divide quickly to prevent irreversible complications that will be prevented by tumor (based on all physiological factors, and bio-Chemistry, and increasse logical thinking of different organ systems, and wide variations of why tumors occur in different organs or places.

إذا كان خلال السرطان ترفع حموضة الدم via warburg notes of their metabolism (explain it excuse me)

lactic acidosis
لمنع الخلايا المناعية من قتلها

inhibition of immune cells to kill cancer cells

فكيف أن الخلايا المناعية تفعل نفس الشيء لكي تنشط؟

ولو آليات تفعيل الورم طبيعية بهدف البقاء، فلماذا
قد تضر ومتى لا تضر ولماذا تقتلها بالكيماوي؟

Core Research Inquiry:
Provide an advanced, integrative analysis synthesizing human physiology, organic biochemistry, endocrinology, gastrointestinal (GIT) neurobiology, autonomic neurocircuits, and the central nervous system (CNS) to resolve the following questions:
Biochemical State & Glycation: Since glucose is fully soluble in human plasma, how does it dissolve at the molecular level, how do Advanced Glycation End-products (AGEs) form, and why do clinical reports describe "deposits" or precipitation in diabetic tissues?
The Clinical Paradox of Wound Healing: Why would a patient with persistent hyperglycemia experience healing of diabetic foot ulcers after eliminating refined flour, industrial sugar, pasta, pastries, and factory-farmed chicken, even while continuing to consume carbohydrate-rich whole foods (potatoes, rice, fruit juice, dark chocolate, raw honey, and dates) without pharmacological intervention?
Pancreatic $\beta$-Cell Trajectory: In sustained insulin resistance, does the endocrine pancreas continuously escalate insulin secretion, or does it eventually cease? What experimental, clinical trial, and clamp-measurement evidence confirms this mechanism?
Endogenous vs. Exogenous Insulin Resistance: Why do peripheral tissues fail to respond to high endogenous insulin yet clear glucose in response to exogenous (injected) insulin? How does pharmacological dosing mechanically override cell-autonomous resistance checkpoints?
Multi-System Regulatory Axis: Detail the molecular biochemistry, gene regulatory networks, gut-derived incretins, and autonomic/CNS feedback loops governing these phenomena.

Core Research Inquiry:
Provide an advanced, integrative analysis synthesizing human physiology, organic biochemistry, endocrinology, gastrointestinal (GIT) neurobiology, autonomic neurocircuits, and the central nervous system (CNS) to resolve the following questions:
Biochemical State & Glycation: Since glucose is fully soluble in human plasma, how does it dissolve at the molecular level, how do Advanced Glycation End-products (AGEs) form, and why do clinical reports describe "deposits" or precipitation in diabetic tissues?
The Clinical Paradox of Wound Healing: Why would a patient with persistent hyperglycemia experience healing of diabetic foot ulcers after eliminating refined flour, industrial sugar, pasta, pastries, and factory-farmed chicken, even while continuing to consume carbohydrate-rich whole foods (potatoes, rice, fruit juice, dark chocolate, raw honey, and dates) without pharmacological intervention?
Pancreatic $\beta$-Cell Trajectory: In sustained insulin resistance, does the endocrine pancreas continuously escalate insulin secretion, or does it eventually cease? What experimental, clinical trial, and clamp-measurement evidence confirms this mechanism?
Endogenous vs. Exogenous Insulin Resistance: Why do peripheral tissues fail to respond to high endogenous insulin yet clear glucose in response to exogenous (injected) insulin? How does pharmacological dosing mechanically override cell-autonomous resistance checkpoints?
Multi-System Regulatory Axis: Detail the molecular biochemistry, gene regulatory networks, gut-derived incretins, and autonomic/CNS feedback loops governing these phenomena.